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Table 1 Prevalence of virulence-associated genes in A+B1 (commensal) and B2+D (virulent) E. coli clones isolated from active and non-active coeliac disease patients and healthy children

From: Reduced diversity and increased virulence-gene carriage in intestinal enterobacteria of coeliac children

Virulence factor (gene) Virulent B2 + D clones (%)* Commensal A + B1 clones (%)*
  Active coeliac (n = 23) Non-active coeliac (n = 23) Healthy (n = 21) Active coeliac (n = 17) Non-active coeliac (n = 19) Healthy (n = 19)
Type 1 fimbriae (fim A) 18 (78)a 18 (78)a 10 (48)a 11 (65)a 14 (82)a 16 (84)a
S fimbriae (sfaD/E) 8 (35)a 9 (39)a 8 (38)a 0a 6 (35) b 1 (5) a
P fimbriae (papC) 5 (22)a 9 (39)a 0b 0a 0a 4 (21)a
Dr haemagglutinin (draA) 0a 0a 0a 0a 0a 0a
Haemolysin (hlyA) 5 (22)ab 9 (39)b 1 (5)a 0a 1 (6)a 4 (21)a
K1 (neuB) 8 (35)a 5 (22)a 5 (24)a 0a 10 (59) b 8 (42)b
K5 (KfiC) 6 (26)a 0b 2 (10)ab 0a 0a 1 (5)a
Aerobactin (iutA) 4 (17)a 4 (17)a 7 (33)a 1 (6)a 6 (35)ab 12 (63) b
  1. *Different letters (a to b) in the same row and within the same phylogenetic group (virulent B2+D or commensal A+B1) indicate significant differences in the gene-carriage.